@article {cite-key,
	title = {Beta-catenin: a key mediator of Wnt signaling.},
	journal = {Curr Opin Genet Dev},
	volume = {8},
	number = {1},
	year = {1998},
	month = {Feb},
	pages = {95{\textendash}102},
	address = {Howard Hughes Medical Institute, Department of Developmental Biology, Backman Center, Stanford University Medical Center, California 94305, USA. willert@cmgm.stanford.edu},
	abstract = {Beta-catenin is a pivotal player in the signaling pathway initiated by Wnt proteins, mediators of several developmental processes. beta-catenin activity is controlled by a large number of binding partners that affect the stability and the localization of beta-catenin and is thereby able to participate in such varying processes as gene expression and cell adhesion. Activating mutations in beta-catenin and in components regulating its stability can contribute to the formation of certain tumors.},
	issn = {0959-437X (Print); 0959-437X (Linking)},
	author = {Willert, K and Nusse, R}
}
